Molecular Biology Reagents

10-Deacetylbaccatin-III is an intermediate for taxol analog preparations. Taxols have exhibit antitumor agents. Several of these taxols can be synthesized from 10- Deacetylbaccatin-III.An intermediate for taxol analog preparations.

10-Deacetylbaccatin-III is an intermediate for taxol analog preparations. Taxols have exhibit antitumor agents. Several of these taxols can be synthesized from 10- Deacetylbaccatin-III.An intermediate for taxol analog preparations.

10-Deacetylbaccatin-III is an intermediate for taxol analog preparations. Taxols have exhibit antitumor agents. Several of these taxols can be synthesized from 10- Deacetylbaccatin-III.An intermediate for taxol analog preparations.

10-Deacetylbaccatin-III is an intermediate for taxol analog preparations. Taxols have exhibit antitumor agents. Several of these taxols can be synthesized from 10- Deacetylbaccatin-III.An intermediate for taxol analog preparations.

10-Formyl-5,8-dideazafolic acid is a thymidylate synthase inhibitorForm:SolidIC50&amp: Target:thymidylate synthase.

10-Formyl-5,8-dideazafolic acid is a thymidylate synthase inhibitorForm:SolidIC50&amp: Target:thymidylate synthase.

10-Formyl-5,8-dideazafolic acid is a thymidylate synthase inhibitorForm:SolidIC50&amp: Target:thymidylate synthase.

DescriptionNatural product derived from plant source.}.

10-OBn-ginkgolide B.

Application 10058-F4 has been used: as c-Myc inhibitor to treat stromal cells as c-Myc inhibitor to determine the effect of c-Myc inhibition on cardiac progenitor cells (CPC) growth as c-Myc inhibitor to culture T cells to treat C4-2 cells to

Application 10058-F4 has been used: as c-Myc inhibitor to treat stromal cells as c-Myc inhibitor to determine the effect of c-Myc inhibition on cardiac progenitor cells (CPC) growth as c-Myc inhibitor to culture T cells to treat C4-2 cells to

A cell-permeable inhibitor of the dimerization of c-Myc and Max at 64 &microM, preventing c-Myc-dependent gene expression and cell proliferation induces cell cycle arrest, apoptosis, and myeloid differentiation at 100 &microM in human acute