Lipids

(±)12-HETE is one of the six monohydroxy fatty acids produced by the non-enzymatic oxidation of arachidonic acid. The biological activity of (±)12-HETE is similar to that of its constituent enantiomers. It aggregates neutrophils with an EC50 value

One of the six monohydroperoxy fatty acids produced by the non-enzymatic oxidation of arachidonic acid and consists of an equivalent mixture of the R and S isomers. Reduction of the hydroperoxide yields the more stable hydroxyl fatty acid

One of the six monohydroperoxy fatty acids produced by the non-enzymatic oxidation of arachidonic acid and consists of an equivalent mixture of the R and S isomers. Reduction of the hydroperoxide yields the more stable hydroxyl fatty acid

(±)13-HDoHE is an autoxidation product of docosahexaenoic acid (DHA) in vitro. Also, it is produced from incubations of DHA in rat brain, intestinal microsomes and liver. It is a potential marker of oxidative stress in brain and retina where DHA is

(±)13-HDoHE is an autoxidation product of docosahexaenoic acid (DHA) in vitro. Also, it is produced from incubations of DHA in rat brain, intestinal microsomes and liver. It is a potential marker of oxidative stress in brain and retina where DHA is

(±)13-HODE is one of the two racemic monohydroxy fatty acids resulting from the non-enzymatic oxidation of linoleic acid. It is the principle hydroxylated fatty acid in human psoriatic skin scales, with a mean concentration of 17 ng/mg.

(±)13-HODE is one of the two racemic monohydroxy fatty acids resulting from the non-enzymatic oxidation of linoleic acid. It is the principle hydroxylated fatty acid in human psoriatic skin scales, with a mean concentration of 17 ng/mg.

(±)13-HODE cholesteryl ester was originally extracted from atherosclerotic lesions and shown to be produced by Cu|2+|-catalyzed oxidation of LDL. Later studies showed 15-LO from rabbit reticulocytes and human monocytes were able to metabolize

(±)13-HODE cholesteryl ester was originally extracted from atherosclerotic lesions and shown to be produced by Cu|2+|-catalyzed oxidation of LDL. Later studies showed 15-LO from rabbit reticulocytes and human monocytes were able to metabolize

14(15)-EET-SI is the methyl sulfonamide analog of 14(15)-EET . It is more metabolically stable than the parent compound because it is no longer sensitive to β-oxidation or membrane esterification. 14(15)-EET-SI stimulates tyrosine phosphorylation

14(15)-EET-SI is the methyl sulfonamide analog of 14(15)-EET . It is more metabolically stable than the parent compound because it is no longer sensitive to β-oxidation or membrane esterification. 14(15)-EET-SI stimulates tyrosine phosphorylation

Produced by the non-enzymatic oxidation of 11,14-eicosadienoic acid, yet there are no reports of biological activity associated with (±)15-HEDE.