Life Science

A methyl ester version of 17(R)-RvD1 which may act as a lipophilic prodrug form that will alter its distribution and pharmacokinetic properties.

A methyl ester version of 17(R)-RvD1 which may act as a lipophilic prodrug form that will alter its distribution and pharmacokinetic properties.

A methyl ester version of 17(R)-RvD1 which may act as a lipophilic prodrug form that will alter its distribution and pharmacokinetic properties.

An aspirin-triggered epimer of RvD1 reduces human leukocyte transendothelial migration (EC50 = ~30 nM), and limits leukocyte infiltration in a mouse model of peritonitis.

An aspirin-triggered epimer of RvD1 reduces human leukocyte transendothelial migration (EC50 = ~30 nM), and limits leukocyte infiltration in a mouse model of peritonitis.

An aspirin-triggered epimer of RvD1 reduces human leukocyte transendothelial migration (EC50 = ~30 nM), and limits leukocyte infiltration in a mouse model of peritonitis.

An aspirin-triggered epimer of RvD1 reduces human leukocyte transendothelial migration (EC50 = ~30 nM), and limits leukocyte infiltration in a mouse model of peritonitis.

An internal standard for the quantification of 17(R)-RvD1 by GC- or LC-MS.

An internal standard for the quantification of 17(R)-RvD1 by GC- or LC-MS.

An internal standard for the quantification of 17(R)-RvD1 by GC- or LC-MS.

An internal standard for the quantification of 17(R)-RvD1 by GC- or LC-MS.

A primary mono-oxygenation product of DHA in human whole blood, human leukocytes, and mouse brain serves as a precursor to 17(S)-resolvins.