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10007466-100Arachidonic acid-biotin was designed to allow arachidonic acid to be detected in complexes with protein binding partners such as fatty acid binding proteins (FABPs). It is thus a tool to be used in the general elucidation of the signaling and
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10007466-500Arachidonic acid-biotin was designed to allow arachidonic acid to be detected in complexes with protein binding partners such as fatty acid binding proteins (FABPs). It is thus a tool to be used in the general elucidation of the signaling and
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10006758-1An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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10006758-100An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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10006758-500An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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390010-1An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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390010-10An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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390010-5An internal standard for the quantification of arachidonic acid by GC- or LC-MS.
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91054-10A potent, stable, and selective agonist analog of AEA with a Ki value of 1.4 nM at the isolated rat CB1 receptor 1,400 times more potent at the CB1 compared with the CB2 receptor induces hypothermia in mice with the same efficacy as AEA, in spite
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91054-25A potent, stable, and selective agonist analog of AEA with a Ki value of 1.4 nM at the isolated rat CB1 receptor 1,400 times more potent at the CB1 compared with the CB2 receptor induces hypothermia in mice with the same efficacy as AEA, in spite
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91054-5A potent, stable, and selective agonist analog of AEA with a Ki value of 1.4 nM at the isolated rat CB1 receptor 1,400 times more potent at the CB1 compared with the CB2 receptor induces hypothermia in mice with the same efficacy as AEA, in spite
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91054-50A potent, stable, and selective agonist analog of AEA with a Ki value of 1.4 nM at the isolated rat CB1 receptor 1,400 times more potent at the CB1 compared with the CB2 receptor induces hypothermia in mice with the same efficacy as AEA, in spite