Life Science

Balofloxacin is a fluoroquinolone antibiotic that displays strong antibacterial activity against gram-positive bacteria and weak antibacterial activity against gram-negative bacteria. Balofloxacin inhibits the supercoiling ability of bacterial DNA

EZ-10 Spin Column Bacterial Genomic DNA Miniprep Kit: EZ-10 Spin Column Bacterial Genomic DNA Miniprep Kit EZ-10 Spin Column Kits provide a fast, simple and efficient method for purification of genomic DNA from various sources such as Bacteria,

PPAR&gamma is a ligand-activated transcription factor involved in the regulation of lipid homeostasis and may function as a master regulator of adipogenesis. Ligands for PPAR&gamma include antidiabetic drugs of the thiazolidinedione structural

PPAR&gamma is a ligand-activated transcription factor involved in the regulation of lipid homeostasis and may function as a master regulator of adipogenesis. Ligands for PPAR&gamma include antidiabetic drugs of the thiazolidinedione structural

PPAR&gamma is a ligand-activated transcription factor involved in the regulation of lipid homeostasis and may function as a master regulator of adipogenesis. Ligands for PPAR&gamma include antidiabetic drugs of the thiazolidinedione structural

PPAR&gamma is a ligand-activated transcription factor involved in the regulation of lipid homeostasis and may function as a master regulator of adipogenesis. Ligands for PPAR&gamma include antidiabetic drugs of the thiazolidinedione structural

A Bcr-Abl kinase inhibitor (IC₅₀ = 5.8 nM) that is 25- to 55-fold more potent than imatinib in vitro and &ge10-fold more potent in vivo inhibits 12 out of the 13 most frequent imatinib-resistant Bcr-Abl point mutations, but not the

A Bcr-Abl kinase inhibitor (IC₅₀ = 5.8 nM) that is 25- to 55-fold more potent than imatinib in vitro and &ge10-fold more potent in vivo inhibits 12 out of the 13 most frequent imatinib-resistant Bcr-Abl point mutations, but not the

A Bcr-Abl kinase inhibitor (IC₅₀ = 5.8 nM) that is 25- to 55-fold more potent than imatinib in vitro and &ge10-fold more potent in vivo inhibits 12 out of the 13 most frequent imatinib-resistant Bcr-Abl point mutations, but not the

B-cell activating factor, also known as BAFF, TALL-1, TNAK, and zTNF4, is a member of theTNF ligand superfamily designated TNFSF13B. Produced by macrophages, dendritic cells, and T lymphocytes, BAFF promotes the survival of B cells and is essential

B-cell activating factor, also known as BAFF, TALL-1, TNAK, and zTNF4, is a member of theTNF ligand superfamily designated TNFSF13B. Produced by macrophages, dendritic cells, and T lymphocytes, BAFF promotes the survival of B cells and is essential

B-cell activating factor, also known as BAFF, TALL-1, TNAK, and zTNF4, is a member of theTNF ligand superfamily designated TNFSF13B. Produced by macrophages, dendritic cells, and T lymphocytes, BAFF promotes the survival of B cells and is essential