Life Science

A SSRI which is commonly prescribed as an antidepressant also acts as an antagonist of nicotinic acetylcholine receptors (IC₅₀= 0.93 &microM).

A SSRI which is commonly prescribed as an antidepressant also acts as an antagonist of nicotinic acetylcholine receptors (IC₅₀= 0.93 &microM).

An agonist of human CAR (EC₅₀ = 49 nM) induces nuclear translocation of human CAR in hepatocytes, followed by increased expression of CAR-regulated genes.

An agonist of human CAR (EC₅₀ = 49 nM) induces nuclear translocation of human CAR in hepatocytes, followed by increased expression of CAR-regulated genes.

An agonist of human CAR (EC₅₀ = 49 nM) induces nuclear translocation of human CAR in hepatocytes, followed by increased expression of CAR-regulated genes.

An endogenous intermediate in the synthesis of phosphatidylcholine that demonstrates protective effects in cerebral ischemia, traumatic brain injury, and memory disorders when administered exogenously.

An endogenous intermediate in the synthesis of phosphatidylcholine that demonstrates protective effects in cerebral ischemia, traumatic brain injury, and memory disorders when administered exogenously.

An endogenous intermediate in the synthesis of phosphatidylcholine that demonstrates protective effects in cerebral ischemia, traumatic brain injury, and memory disorders when administered exogenously.

An endogenous intermediate in the synthesis of phosphatidylcholine that demonstrates protective effects in cerebral ischemia, traumatic brain injury, and memory disorders when administered exogenously.

A mycotoxin that inhibits the mitochondrial ATP synthetase system inhibits soluble ATPase (KD = 4.1 &microM), ADP-stimulated respiration in isolated rat liver mitochondria (KD = 0.

A mycotoxin that inhibits the mitochondrial ATP synthetase system inhibits soluble ATPase (KD = 4.1 &microM), ADP-stimulated respiration in isolated rat liver mitochondria (KD = 0.

A mycotoxin found as food contaminants that causes mycotoxic nephropathy in livestock and Balkan nephropathy and yellow rice fever in humans induces apoptosis, blocks tubulin polymerization, and disrupts mitotic spindle assembly.