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11320-10A mycotoxin found as food contaminants that causes mycotoxic nephropathy in livestock and Balkan nephropathy and yellow rice fever in humans induces apoptosis, blocks tubulin polymerization, and disrupts mitotic spindle assembly.
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11320-5A mycotoxin found as food contaminants that causes mycotoxic nephropathy in livestock and Balkan nephropathy and yellow rice fever in humans induces apoptosis, blocks tubulin polymerization, and disrupts mitotic spindle assembly.
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16172-1A highly sensitive, rhodamine phenylglyoxal-based fluorophore that specifically detects protein citrullination via a chemoselective reaction between glyoxal and citrulline reacts with any citrulline-containing protein and can be analyzed with
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16172-250A highly sensitive, rhodamine phenylglyoxal-based fluorophore that specifically detects protein citrullination via a chemoselective reaction between glyoxal and citrulline reacts with any citrulline-containing protein and can be analyzed with
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16172-500A highly sensitive, rhodamine phenylglyoxal-based fluorophore that specifically detects protein citrullination via a chemoselective reaction between glyoxal and citrulline reacts with any citrulline-containing protein and can be analyzed with
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10010355-1A potent and selective antagonist of the EP4 receptor (Ki = 13 and 20 nM for human and rat EP4, respectively) inhibits PGE2-evoked elevation in intracellular cAMP in cells and, in vivo, reduces thermal hyperalgesia induced by intraplantar injection
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10010355-10A potent and selective antagonist of the EP4 receptor (Ki = 13 and 20 nM for human and rat EP4, respectively) inhibits PGE2-evoked elevation in intracellular cAMP in cells and, in vivo, reduces thermal hyperalgesia induced by intraplantar injection
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10010355-25A potent and selective antagonist of the EP4 receptor (Ki = 13 and 20 nM for human and rat EP4, respectively) inhibits PGE2-evoked elevation in intracellular cAMP in cells and, in vivo, reduces thermal hyperalgesia induced by intraplantar injection
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10010355-5A potent and selective antagonist of the EP4 receptor (Ki = 13 and 20 nM for human and rat EP4, respectively) inhibits PGE2-evoked elevation in intracellular cAMP in cells and, in vivo, reduces thermal hyperalgesia induced by intraplantar injection
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10010428-10A selective antagonist of EP4 (Ki = 3.2 nM) that less potently binds EP2 (Ki = 630 nM) and has no affinity for EP1 or EP3 delays the healing of gastric ulcers, suppressing the upregulation of VEGF expression and angiogenesis blocks pain and
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10010428-100A selective antagonist of EP4 (Ki = 3.2 nM) that less potently binds EP2 (Ki = 630 nM) and has no affinity for EP1 or EP3 delays the healing of gastric ulcers, suppressing the upregulation of VEGF expression and angiogenesis blocks pain and
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10010428-250A selective antagonist of EP4 (Ki = 3.2 nM) that less potently binds EP2 (Ki = 630 nM) and has no affinity for EP1 or EP3 delays the healing of gastric ulcers, suppressing the upregulation of VEGF expression and angiogenesis blocks pain and