Life Science

Bisdemethoxycurcumin, like its parent compound curcumin, exhibits anticancer, antioxidative, anti-diabetic, anti-inflammatory, and antibacterial activities. Bisdemethoxycurcumin inhibits Wnt signaling by inhibiting WIF-1 promoter demethylation and

Bisdemethoxycurcumin, like its parent compound curcumin, exhibits anticancer, antioxidative, anti-diabetic, anti-inflammatory, and antibacterial activities. Bisdemethoxycurcumin inhibits Wnt signaling by inhibiting WIF-1 promoter demethylation and

Bisdemethoxycurcumin, like its parent compound curcumin, exhibits anticancer, antioxidative, anti-diabetic, anti-inflammatory, and antibacterial activities. Bisdemethoxycurcumin inhibits Wnt signaling by inhibiting WIF-1 promoter demethylation and

A natural product with antioxidant, anti-tumor, and anti-inflammatory properties.

A natural product with antioxidant, anti-tumor, and anti-inflammatory properties.

A natural product with antioxidant, anti-tumor, and anti-inflammatory properties.

A small molecule upregulator of SMN that has been shown in vitro to increase levels of SMN in SMA-patient fibroblasts by 50% at 18 &microM increases phosphorylation of ERK to initiate Ras-Raf-MEK signaling, which results in an increased rate of

A small molecule upregulator of SMN that has been shown in vitro to increase levels of SMN in SMA-patient fibroblasts by 50% at 18 &microM increases phosphorylation of ERK to initiate Ras-Raf-MEK signaling, which results in an increased rate of

A small molecule upregulator of SMN that has been shown in vitro to increase levels of SMN in SMA-patient fibroblasts by 50% at 18 &microM increases phosphorylation of ERK to initiate Ras-Raf-MEK signaling, which results in an increased rate of

A small molecule upregulator of SMN that has been shown in vitro to increase levels of SMN in SMA-patient fibroblasts by 50% at 18 &microM increases phosphorylation of ERK to initiate Ras-Raf-MEK signaling, which results in an increased rate of

A potent antagonist of the PAF receptor, inhibiting aggregation of rabbit and human platelets induced by PAF with IC₅₀ values of 75 and 170 nM, respectively prevents PAF-induced hypotension in rats, while not blocking hypotension

A potent antagonist of the PAF receptor, inhibiting aggregation of rabbit and human platelets induced by PAF with IC₅₀ values of 75 and 170 nM, respectively prevents PAF-induced hypotension in rats, while not blocking hypotension