Biologically Active Small Molecules

10-Pyrene-PC is a substrate for all PLA2s with the exception of cPLA2 and PAF-AH. Upon phospholipid hydrolysis, 10-pyrenyldecanoic acid is produced from 10-pyrene-PC.

10-Pyrene-PC is a substrate for all PLA2s with the exception of cPLA2 and PAF-AH. Upon phospholipid hydrolysis, 10-pyrenyldecanoic acid is produced from 10-pyrene-PC.

10-Pyrene-PC is a substrate for all PLA2s with the exception of cPLA2 and PAF-AH. Upon phospholipid hydrolysis, 10-pyrenyldecanoic acid is produced from 10-pyrene-PC.

A FAHFA in which stearic acid is esterified to 10-hydroxy stearic acid.

A FAHFA in which stearic acid is esterified to 10-hydroxy stearic acid.

A FAHFA in which stearic acid is esterified to 10-hydroxy stearic acid.

Heteroatom-substituted fatty acids have been observed to modulate the extension and desaturating of fatty acids, and to influence their distribution within phospholipids pools. 10-Thiastearic acid inhibits desaturation of radiolabeled stearate to

Heteroatom-substituted fatty acids have been observed to modulate the extension and desaturating of fatty acids, and to influence their distribution within phospholipids pools. 10-Thiastearic acid inhibits desaturation of radiolabeled stearate to

Heteroatom-substituted fatty acids have been observed to modulate the extension and desaturating of fatty acids, and to influence their distribution within phospholipids pools. 10-Thiastearic acid inhibits desaturation of radiolabeled stearate to

Heteroatom-substituted fatty acids have been observed to modulate the extension and desaturating of fatty acids, and to influence their distribution within phospholipids pools. 10-Thiastearic acid inhibits desaturation of radiolabeled stearate to

A cell-permeable inhibitor of the dimerization of c-Myc and Max at 64 &microM, preventing c-Myc-dependent gene expression and cell proliferation induces cell cycle arrest, apoptosis, and myeloid differentiation at 100 &microM in human acute

A cell-permeable inhibitor of the dimerization of c-Myc and Max at 64 &microM, preventing c-Myc-dependent gene expression and cell proliferation induces cell cycle arrest, apoptosis, and myeloid differentiation at 100 &microM in human acute