Biologically Active Small Molecules

8-piperazin-1-yl-Isoquinoline (hydrochloride) is a synthetic intermediate useful for pharmaceutical synthesis.

A prenylflavonoid with potent estrogenic activity that inhibits both ER&alpha and ER&beta (IC₅₀s = 57 and 68 nM, respectively) effective in vivo, suppressing bone loss and temperature changes in rat models of osteoporosis and

A prenylflavonoid with potent estrogenic activity that inhibits both ER&alpha and ER&beta (IC₅₀s = 57 and 68 nM, respectively) effective in vivo, suppressing bone loss and temperature changes in rat models of osteoporosis and

A prenylflavonoid with potent estrogenic activity that inhibits both ER&alpha and ER&beta (IC₅₀s = 57 and 68 nM, respectively) effective in vivo, suppressing bone loss and temperature changes in rat models of osteoporosis and

A prenylflavonoid with potent estrogenic activity that inhibits both ER&alpha and ER&beta (IC₅₀s = 57 and 68 nM, respectively) effective in vivo, suppressing bone loss and temperature changes in rat models of osteoporosis and

CLA refers to a family of 8 geometric isomers of linoleic acid in which the two double bonds are contiguous. 9(E),11(E)-CLA is the 9,11 all-trans isomer of linoleic acid.

9(R)-HETE is an enantiomer which makes up 50% of (&plusmn)9-HETE (Item No. 34400). At a concentration of 300 nM, 9(R)-HETE activates RXR&gamma-dependent transcription 1.5 fold relative to a control.

9(R)-HETE is an enantiomer which makes up 50% of (&plusmn)9-HETE (Item No. 34400). At a concentration of 300 nM, 9(R)-HETE activates RXR&gamma-dependent transcription 1.5 fold relative to a control.

9(R)-HETE is an enantiomer which makes up 50% of (&plusmn)9-HETE (Item No. 34400). At a concentration of 300 nM, 9(R)-HETE activates RXR&gamma-dependent transcription 1.5 fold relative to a control.

9(R)-HODE cholesteryl ester was originally extracted from atherosclerotic lesions. It remains uncertain whether the oxidized fatty acid portion of the molecule results from enzymatic lipoxygenation or from random lipid peroxidation.

9(R)-HODE cholesteryl ester was originally extracted from atherosclerotic lesions. It remains uncertain whether the oxidized fatty acid portion of the molecule results from enzymatic lipoxygenation or from random lipid peroxidation.

9(R)-HODE cholesteryl ester was originally extracted from atherosclerotic lesions. It remains uncertain whether the oxidized fatty acid portion of the molecule results from enzymatic lipoxygenation or from random lipid peroxidation.