Biologically Active Small Molecules

A diterpenoid compound that inhibits NF-&kappaB binding to DNA promoters and induces expression of the CYP1A enzymes to produce immunosuppressant, antithrombotic, anti-inflammatory, antineoplastic, antiviral, antibacterial, antidiabetic,

A diterpenoid compound that inhibits NF-&kappaB binding to DNA promoters and induces expression of the CYP1A enzymes to produce immunosuppressant, antithrombotic, anti-inflammatory, antineoplastic, antiviral, antibacterial, antidiabetic,

A diterpenoid compound that inhibits NF-&kappaB binding to DNA promoters and induces expression of the CYP1A enzymes to produce immunosuppressant, antithrombotic, anti-inflammatory, antineoplastic, antiviral, antibacterial, antidiabetic,

A diterpenoid compound that inhibits NF-&kappaB binding to DNA promoters and induces expression of the CYP1A enzymes to produce immunosuppressant, antithrombotic, anti-inflammatory, antineoplastic, antiviral, antibacterial, antidiabetic,

A steroid hormone produced in the body from 5&alpha-reduced metabolites of dehydroepiandrosterone that possesses 7-fold weaker androgen potency compared to that of testosterone excreted in urine and used as the international reference standard for

A steroid hormone produced in the body from 5&alpha-reduced metabolites of dehydroepiandrosterone that possesses 7-fold weaker androgen potency compared to that of testosterone excreted in urine and used as the international reference standard for

A steroid hormone produced in the body from 5&alpha-reduced metabolites of dehydroepiandrosterone that possesses 7-fold weaker androgen potency compared to that of testosterone excreted in urine and used as the international reference standard for

A steroid hormone produced in the body from 5&alpha-reduced metabolites of dehydroepiandrosterone that possesses 7-fold weaker androgen potency compared to that of testosterone excreted in urine and used as the international reference standard for

A classical nitroxyl (NO-) donor dissociates in a pH-dependent, first order process with a half-life of 2.3 minutes at 37&degC, pH 7.4, to liberate 0.54 moles for NO per mole of parent compound.

A classical nitroxyl (NO-) donor dissociates in a pH-dependent, first order process with a half-life of 2.3 minutes at 37&degC, pH 7.4, to liberate 0.54 moles for NO per mole of parent compound.

A classical nitroxyl (NO-) donor dissociates in a pH-dependent, first order process with a half-life of 2.3 minutes at 37&degC, pH 7.4, to liberate 0.54 moles for NO per mole of parent compound.

A classical nitroxyl (NO-) donor dissociates in a pH-dependent, first order process with a half-life of 2.3 minutes at 37&degC, pH 7.4, to liberate 0.54 moles for NO per mole of parent compound.