Biologically Active Small Molecules

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor in vivo activity is enhanced much less than the binding affinity,

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor in vivo activity is enhanced much less than the binding affinity,

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor in vivo activity is enhanced much less than the binding affinity,

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor in vivo activity is enhanced much less than the binding affinity,

An analog of AEA that exhibits weak or no binding to the human CB1 receptor (Ki &gt1 &microM) inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that exhibits weak or no binding to the human CB1 receptor (Ki &gt1 &microM) inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that exhibits weak or no binding to the human CB1 receptor (Ki &gt1 &microM) inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that exhibits weak or no binding to the human CB1 receptor (Ki &gt1 &microM) inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that binds to the human CB1 receptor with a Ki value of 60 nM inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that binds to the human CB1 receptor with a Ki value of 60 nM inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that binds to the human CB1 receptor with a Ki value of 60 nM inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.

An analog of AEA that binds to the human CB1 receptor with a Ki value of 60 nM inhibits rat glial gap junction cell-cell communication at a concentration of 50 &microM.