Biologically Active Small Molecules

Features Each vial contains a mixture of standards packaged in amber ampules purged with argon &gt1 ml provided at the concentration specified in the insert Ideally suited for use as an internal standard in mass spectrometry applications This

Features Each vial contains a mixture of standards packaged in amber ampules purged with argon &gt1 ml provided at the concentration specified in the insert Ideally suited for use as an internal standard in mass spectrometry applications The

A synthetic glucocorticoid that binds the human glucocorticoid receptor with a higher affinity than a natural ligand, cortisol (Kd = 5 vs. 17 nM, respectively) has anti-inflammatory effects.

A synthetic glucocorticoid that binds the human glucocorticoid receptor with a higher affinity than a natural ligand, cortisol (Kd = 5 vs. 17 nM, respectively) has anti-inflammatory effects.

A synthetic glucocorticoid that binds the human glucocorticoid receptor with a higher affinity than a natural ligand, cortisol (Kd = 5 vs. 17 nM, respectively) has anti-inflammatory effects.

The (S)-enantiomer of medetomidine, an imidazole compound that binds to &alpha2-adrenoceptors (Ki = 1.08 nM) with high affinity and selectivity its activation of &alpha2-adrenoceptors produces analgesic, sedative, and anxiolytic effects.

The (S)-enantiomer of medetomidine, an imidazole compound that binds to &alpha2-adrenoceptors (Ki = 1.08 nM) with high affinity and selectivity its activation of &alpha2-adrenoceptors produces analgesic, sedative, and anxiolytic effects.

The (S)-enantiomer of medetomidine, an imidazole compound that binds to &alpha2-adrenoceptors (Ki = 1.08 nM) with high affinity and selectivity its activation of &alpha2-adrenoceptors produces analgesic, sedative, and anxiolytic effects.

The (S)-enantiomer of medetomidine, an imidazole compound that binds to &alpha2-adrenoceptors (Ki = 1.08 nM) with high affinity and selectivity its activation of &alpha2-adrenoceptors produces analgesic, sedative, and anxiolytic effects.

An intracellular iron chelator and an inhibitor of topoisomerase II&alpha that is highly protective in reducing anthracycline-induced cardiotoxicity and extravasation injury scavenges hydroxyl (IC₅₀ = 2.1 mM), superoxide

An intracellular iron chelator and an inhibitor of topoisomerase II&alpha that is highly protective in reducing anthracycline-induced cardiotoxicity and extravasation injury scavenges hydroxyl (IC₅₀ = 2.1 mM), superoxide

An intracellular iron chelator and an inhibitor of topoisomerase II&alpha that is highly protective in reducing anthracycline-induced cardiotoxicity and extravasation injury scavenges hydroxyl (IC₅₀ = 2.1 mM), superoxide