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B646620-5mgBevenopran is a peripheral μ-opioid receptor antagonistIn VivoBevenopran is a peripheral μ-opioid receptor antagonist. Bevenopran is currently under investigation for the treatment of opioid-induced bowel dysfunction (OBD) . Bevenopran tends to
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B654661-1mlBevenopran is a peripheral μ-opioid receptor antagonistIn VivoBevenopran is a peripheral μ-opioid receptor antagonist. Bevenopran is currently under investigation for the treatment of opioid-induced bowel dysfunction (OBD) . Bevenopran tends to
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B651666-10mgBGC-20-1531 (PGN 1531) free base is a potent and selective prostanoid EP 4 receptor antagonist, with a pK B of 7.6. BGC-20-1531 free base has the potential for the research of migraine headacheIn VitroBGC-20-1531 free base competitively antagonized
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B651666-25mgBGC-20-1531 (PGN 1531) free base is a potent and selective prostanoid EP 4 receptor antagonist, with a pK B of 7.6. BGC-20-1531 free base has the potential for the research of migraine headacheIn VitroBGC-20-1531 free base competitively antagonized
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B651666-5mgBGC-20-1531 (PGN 1531) free base is a potent and selective prostanoid EP 4 receptor antagonist, with a pK B of 7.6. BGC-20-1531 free base has the potential for the research of migraine headacheIn VitroBGC-20-1531 free base competitively antagonized
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B656774-1mlBGC-20-1531 (PGN 1531) free base is a potent and selective prostanoid EP 4 receptor antagonist, with a pK B of 7.6. BGC-20-1531 free base has the potential for the research of migraine headacheIn VitroBGC-20-1531 free base competitively antagonized
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B648906-100mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They
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B648906-10mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They
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B648906-1mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They
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B648906-25mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They
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B648906-50mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They
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B648906-5mgBGG463 (K03859) is an orally active type II CDK2 inhibitorIn VitroBGG463 potently inhibits T315I BCR–ABL autophosphorylation and shows good oral efficacy in mouse models of CML. MCE has not independently confirmed the accuracy of these methods. They