Metabolites

9α,11β-PGF2 (9α,11β-Prostaglandin F2), a prostaglandin metabolite that is enzymatically formed in the liver, lung, and kidneys, has been reported to cause constriction of canine coronary artery strips and inhibit platelet aggregation. Additional

9α,11β-PGF2 (9α,11β-Prostaglandin F2), a prostaglandin metabolite that is enzymatically formed in the liver, lung, and kidneys, has been reported to cause constriction of canine coronary artery strips and inhibit platelet aggregation. Additional

9α,11β-PGF2 (9α,11β-Prostaglandin F2), a prostaglandin metabolite that is enzymatically formed in the liver, lung, and kidneys, has been reported to cause constriction of canine coronary artery strips and inhibit platelet aggregation. Additional

[³⁵S]ATPαS.

[³⁵S]ATPαS.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A 83-01 is a potent inhibitor of TGF-β type I receptor ALK5 kinase, type I nodal receptor ALK4 and type I nodal receptor ALK7, with IC50s of 12 nM, 45 nM and 7.5 nM against the transcription induced by ALK5, ALK4 and ALK7, respectively。.

A2P5P.