Metabolites

1-O-hexadecyl-2-acetyl-sn-glycero-3-phosphocholine.

1-OH-PGE₁.

1-OH-PGE₁.

1-Stearoyl-2-arachidonoyl-sn-glycerol is a diacylglycerol derivative, and an endogenous activator of PKC (protein kinase C). 1-Stearoyl-2-arachidonoyl-sn-glycerol is produced via receptor-mediated inositol phospholipid hydrolysis.

1-Stearoyl-2-arachidonoyl-sn-glycerol is a diacylglycerol derivative, and an endogenous activator of PKC (protein kinase C). 1-Stearoyl-2-arachidonoyl-sn-glycerol is produced via receptor-mediated inositol phospholipid hydrolysis.

1-stearoyl-lysophosphatidylcholine.

1-stearoyl-lysophosphatidylcholine.

10058-F4 is a c-Myc inhibitor that specificallly inhibits the c-Myc-Max interaction and prevents transactivation of c-Myc target gene expression.A c-Myc inhibitor and apoptosis inducer.

11-cis-Retinol is a metabolite of Vitamin A. Retinol and derivatives of retinol such as 11-cis-Retinol play an essential role in metabolic functioning of the retina, the growth of and differentiation of epithelial tissue, the growth of bone,

11-cis-Retinol is a metabolite of Vitamin A. Retinol and derivatives of retinol such as 11-cis-Retinol play an essential role in metabolic functioning of the retina, the growth of and differentiation of epithelial tissue, the growth of bone,

11-Dehydrocorticosterone is a endogenous corticosteroid. 11-Dehydrocorticosterone can be a source of transcriptionally active glucocorticoid in cardiac myocytes and fibroblasts. 11-Dehydrocorticosterone can increase SGK mRNA expression in cardiac

11-Dehydrocorticosterone is a endogenous corticosteroid. 11-Dehydrocorticosterone can be a source of transcriptionally active glucocorticoid in cardiac myocytes and fibroblasts. 11-Dehydrocorticosterone can increase SGK mRNA expression in cardiac