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C608317-5mgcarbinoxamine.
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C355698-100μgCarbocyclic Thromboxane A2(CTA2) is a stable analog of TXA2 which acts as a potent coronary vasoconstrictor at low concentrations. Unlike other vascular TP receptor agonists, CTA2 is a potent inhibitor of prostanoid-induced platelet aggregation,
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C355698-500μgCarbocyclic Thromboxane A2(CTA2) is a stable analog of TXA2 which acts as a potent coronary vasoconstrictor at low concentrations. Unlike other vascular TP receptor agonists, CTA2 is a potent inhibitor of prostanoid-induced platelet aggregation,
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C608318-25mgcarbocyclic thromboxane A2.
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C608318-5mgcarbocyclic thromboxane A2.
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C127870-100mgCarfilzomib (PR-171) is an irreversible proteasome inhibitor with IC50 of <:5 nM, displayed preferential in vitro inhibitory potency against the ChT-L activity in the β5 subunit, but little or no effect on the PGPH and T-L activities.Proteasome
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C127870-10mgCarfilzomib (PR-171) is an irreversible proteasome inhibitor with IC50 of <:5 nM, displayed preferential in vitro inhibitory potency against the ChT-L activity in the β5 subunit, but little or no effect on the PGPH and T-L activities.Proteasome
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C127870-25mgCarfilzomib (PR-171) is an irreversible proteasome inhibitor with IC50 of <:5 nM, displayed preferential in vitro inhibitory potency against the ChT-L activity in the β5 subunit, but little or no effect on the PGPH and T-L activities.Proteasome
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C127870-500mgCarfilzomib (PR-171) is an irreversible proteasome inhibitor with IC50 of <:5 nM, displayed preferential in vitro inhibitory potency against the ChT-L activity in the β5 subunit, but little or no effect on the PGPH and T-L activities.Proteasome
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C127870-50mgCarfilzomib (PR-171) is an irreversible proteasome inhibitor with IC50 of <:5 nM, displayed preferential in vitro inhibitory potency against the ChT-L activity in the β5 subunit, but little or no effect on the PGPH and T-L activities.Proteasome