Wnt family ligands

STAT5-IN-2 is a STAT5 inhibitor,STAT5-IN-2 has potent antileukemic effectIn VitroSTAT5-IN-2 has EC 50 values of 9 μM and 5 μM in K562 and KU812 cells, respectively. STAT5-IN-2 (100 nM-50 µM 48 hours) has EC 50 values of 2.6 µM and 3.5 µM in KG1a

STAT5-IN-2 is a STAT5 inhibitor,STAT5-IN-2 has potent antileukemic effectIn VitroSTAT5-IN-2 has EC 50 values of 9 μM and 5 μM in K562 and KU812 cells, respectively. STAT5-IN-2 (100 nM-50 µM 48 hours) has EC 50 values of 2.6 µM and 3.5 µM in KG1a

STAT5-IN-2 is a STAT5 inhibitor,STAT5-IN-2 has potent antileukemic effectIn VitroSTAT5-IN-2 has EC 50 values of 9 μM and 5 μM in K562 and KU812 cells, respectively. STAT5-IN-2 (100 nM-50 µM 48 hours) has EC 50 values of 2.6 µM and 3.5 µM in KG1a

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

STX-0119 is a selective, orally active STAT3 dimerization inhibitor. STX-0119 inhibits STAT3 transcription with an IC 50 of 74 μMIn VitroSTX-0119 (10-50 μM 24 h) inhibits STAT3 dimerization through a direct interaction with the STAT3 protein and

Sulindac sulfide is a noncompetitive γ-secretase inhibitor, with an IC 50 of 20.2 μM for γ 42 -secretase activity.In VitroTreatment with 100 μM of Sulindac sulfide (SSide) causes cell death presumably by inducing apoptosis, resulting in marked

Sulindac sulfide is a noncompetitive γ-secretase inhibitor, with an IC 50 of 20.2 μM for γ 42 -secretase activity.In VitroTreatment with 100 μM of Sulindac sulfide (SSide) causes cell death presumably by inducing apoptosis, resulting in marked

Sulindac sulfide is a noncompetitive γ-secretase inhibitor, with an IC 50 of 20.2 μM for γ 42 -secretase activity.In VitroTreatment with 100 μM of Sulindac sulfide (SSide) causes cell death presumably by inducing apoptosis, resulting in marked