Adrenergic blocking agent ethanolamines

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor (Ki = 5.7 nM in rat brain) in vivo activity is enhanced much less than

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor (Ki = 5.7 nM in rat brain) in vivo activity is enhanced much less than

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor (Ki = 5.7 nM in rat brain) in vivo activity is enhanced much less than

An analog of AEA in which the alcohol of the ethanolamide group has been replaced with a fluorine atom, which increases the binding affinity and selectivity for the CB1 receptor (Ki = 5.7 nM in rat brain) in vivo activity is enhanced much less than

2-(2-Amino-phenoxy)-ethanol

2-(2-Amino-phenoxy)-ethanol

2-(2-Amino-phenoxy)-ethanol

2-(Bis(pyridin-2-ylmethyl)amino)ethanol

2-(Bis(pyridin-2-ylmethyl)amino)ethanol

2-(Propylamino)ethanol

2-Amino-1-(2,5-dimethoxyphenyl)ethanol induces alpha-adrenergic receptor stimulation of arterial and venous systems without causing direct cardiac effects to the central nervous system.

2-Amino-1-(2,5-dimethoxyphenyl)ethanol induces alpha-adrenergic receptor stimulation of arterial and venous systems without causing direct cardiac effects to the central nervous system.