Ambazone

BMS-986242 is an orally active, potent and selective indoleamine-2,3-dioxygenase 1 ( IDO1 ) inhibitor. BMS-986242 can be used for the research of cancerIn VitroBMS-986242 is more prone to oxidative metabolism and less susceptible to glucuronidation.

BMS-986242 is an orally active, potent and selective indoleamine-2,3-dioxygenase 1 ( IDO1 ) inhibitor. BMS-986242 can be used for the research of cancerIn VitroBMS-986242 is more prone to oxidative metabolism and less susceptible to glucuronidation.

BMS-986242 is an orally active, potent and selective indoleamine-2,3-dioxygenase 1 ( IDO1 ) inhibitor. BMS-986242 can be used for the research of cancerIn VitroBMS-986242 is more prone to oxidative metabolism and less susceptible to glucuronidation.

BMS-986242 is an orally active, potent and selective indoleamine-2,3-dioxygenase 1 ( IDO1 ) inhibitor. BMS-986242 can be used for the research of cancerIn VitroBMS-986242 is more prone to oxidative metabolism and less susceptible to glucuronidation.

Potent and irreversible BMX inhibitor.

Potent and irreversible BMX inhibitor.

Potent and irreversible BMX inhibitor.

A linear imidazoquinazoline that potently and selectively inhibits the tyrosine kinase activity of EGFR (IC₅₀ = 8 pM).

A linear imidazoquinazoline that potently and selectively inhibits the tyrosine kinase activity of EGFR (IC₅₀ = 8 pM).

InformationBTSA1 is a pharmacologically optimizedBAXactivator that binds with high affinity and specificity to the N-terminal activation site and induces conformational changes to BAX leading to BAX-mediated apoptosis. It effectively promotes

InformationBTSA1 is a pharmacologically optimizedBAXactivator that binds with high affinity and specificity to the N-terminal activation site and induces conformational changes to BAX leading to BAX-mediated apoptosis. It effectively promotes

InformationBTSA1 is a pharmacologically optimizedBAXactivator that binds with high affinity and specificity to the N-terminal activation site and induces conformational changes to BAX leading to BAX-mediated apoptosis. It effectively promotes