Capecitabine

Microhelenin C is a sesquiterpene lactone that can be isolated from Helenium microcephalum , with antitumor activity.Form:Solid.

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A salicylanilide compound with antihelminthic actions specifically inhibits STAT3 (IC50 = 0.25 &muM) inhibits the proliferation of Du145 prostate cancer cells, which have constitutively active STAT3 (IC50 = 0.7 &muM).

PKF118-310 is an inhibitor of survivin expression and antagonist of Tcf4/b-catenin signaling. Shown to induce apoptosis in tumor cells.

PKF118-310 is an inhibitor of survivin expression and antagonist of Tcf4/b-catenin signaling. Shown to induce apoptosis in tumor cells.

PKF118-310 is an inhibitor of survivin expression and antagonist of Tcf4/b-catenin signaling. Shown to induce apoptosis in tumor cells.

Capecitabine is an antifolate anticancer chemotherapeutic that acts as a prodrug of 5-fluorouracil (5-FU), a fluoropyrimidine carbamate that inhibits thymidylate synthase. Capecitabine is converted to 5-FU by thymidine phosphorylase in vivo.

Capecitabine is an antifolate anticancer chemotherapeutic that acts as a prodrug of 5-fluorouracil (5-FU), a fluoropyrimidine carbamate that inhibits thymidylate synthase. Capecitabine is converted to 5-FU by thymidine phosphorylase in vivo.

Capecitabine is an antifolate anticancer chemotherapeutic that acts as a prodrug of 5-fluorouracil (5-FU), a fluoropyrimidine carbamate that inhibits thymidylate synthase. Capecitabine is converted to 5-FU by thymidine phosphorylase in vivo.

Wavelength Clear Ultrasound Gel is a medium viscosity, dye free gel. It contains no salt, alcohol or fragrance: advanced water soluble, hypoallergenic, bacteriostatic formulation: conductive, non-corrosive, greaseless & non-staining.

A functionally selective inhibitor of Chk1 (Kd = 2 nM IC₅₀ = 3 nM) interacts synergistically with DNA antimetabolite agents in vitro and in vivo to selectively induce double-strand DNA breaks and cell death in tumor cells.

Flavonoid that is selectively cytotoxic toward cancer cells. Induces mitochondrial ROS, DNA damage, and metabolic suppression, preferentially in tumor cells. Scutellarein has been shown to have better absorption than scutellarin in Caco-2 monolayer