Sorafenib tosylate

Sorafenib Tosylate (Bay 43-9006) is a multikinase inhibitor of Raf-1, B-Raf and VEGFR-2 with IC50 of 6 nM, 22 nM and 90 nM, respectively.An inhibitor of Flk-1 (VEGFR), PDGFR and Raf kinases.

Sorafenib Tosylate (Bay 43-9006) is a multikinase inhibitor of Raf-1, B-Raf and VEGFR-2 with IC50 of 6 nM, 22 nM and 90 nM, respectively.An inhibitor of Flk-1 (VEGFR), PDGFR and Raf kinases.

Sorafenib Tosylate (Bay 43-9006) is a multikinase inhibitor of Raf-1, B-Raf and VEGFR-2 with IC50 of 6 nM, 22 nM and 90 nM, respectively.An inhibitor of Flk-1 (VEGFR), PDGFR and Raf kinases.

Sorafenib Tosylate (Bay 43-9006) is a multikinase inhibitor of Raf-1, B-Raf and VEGFR-2 with IC50 of 6 nM, 22 nM and 90 nM, respectively.An inhibitor of Flk-1 (VEGFR), PDGFR and Raf kinases.

InformationSotrastaurin (AEB071) Sotrastaurin (AEB071) is a potent and selective pan- PKC inhibitor, mostly for PKCθ with K i of 0.22 nM in a cell-free assay: inactive to PKCζ. Phase 2.In vitroSotrastaurin (&lt: 10μM) treatment effectively abrogated

General description Human Sphingosine kinase 2 (GenBank Accession No. AF245447), full length with N-terminal His tag, MW = 69.

T-1101 tosylate (TAI-95 tosylate) is a Hec1/Nek2 inhibitor with anti-tumor activity. T-1101 tosylate has no inhibitory effect on normal cells, kinases and hERG.

T-1101 tosylate (TAI-95 tosylate) is a Hec1/Nek2 inhibitor with anti-tumor activity. T-1101 tosylate has no inhibitory effect on normal cells, kinases and hERG.

T-1101 tosylate (TAI-95 tosylate) is a Hec1/Nek2 inhibitor with anti-tumor activity. T-1101 tosylate has no inhibitory effect on normal cells, kinases and hERG.

T-1101 tosylate (TAI-95 tosylate) is a Hec1/Nek2 inhibitor with anti-tumor activity. T-1101 tosylate has no inhibitory effect on normal cells, kinases and hERG.

T-1101 tosylate (TAI-95 tosylate) is a Hec1/Nek2 inhibitor with anti-tumor activity. T-1101 tosylate has no inhibitory effect on normal cells, kinases and hERG.

A small molecule, multi-targeted receptor tyrosine kinase inhibitor blocks the autophosphorylation of both wild type and mutant forms of Kit in response to stem cell factor, resulting in cell death active in vivo and commonly used against solid