Zuclopenthixol acetate

A potent, selective, brain permeable inhibitor of GSK3&alpha and GSK3&beta (Kis = 6.9 and 31 nM, respectively) shows good bioavailability after oral administration in vivo, inhibiting hippocampal tau phosphorylation and reversing cognitive

A potent, selective, brain permeable inhibitor of GSK3&alpha and GSK3&beta (Kis = 6.9 and 31 nM, respectively) shows good bioavailability after oral administration in vivo, inhibiting hippocampal tau phosphorylation and reversing cognitive

A potent, selective, brain permeable inhibitor of GSK3&alpha and GSK3&beta (Kis = 6.9 and 31 nM, respectively) shows good bioavailability after oral administration in vivo, inhibiting hippocampal tau phosphorylation and reversing cognitive

DescriptionThe CdS/ZnS or CdSe/ZnS quantum dots functionalized with PEG exhibit low non-specific binding which makes them suitable for a variety of in vivo and in vitro biomedical applications.

DescriptionReactive group: None.The CdS/ZnS or CdSe/ZnS quantum dots functionalized with PEG exhibit low non-specific binding which makes them suitable for a variety of in vivo and in vitro biomedical applications.

DescriptionReactive group: None.The CdS/ZnS or CdSe/ZnS quantum dots functionalized with PEG exhibit low non-specific binding which makes them suitable for a variety of in vivo and in vitro biomedical applications.

DescriptionReactive group: None.The CdS/ZnS or CdSe/ZnS quantum dots functionalized with PEG exhibit low non-specific binding which makes them suitable for a variety of in vivo and in vitro biomedical applications.

Cu(II)GTSM, a cell-permeable Cu-complex, significantly inhibits GSK3β. Cu(II)GTSM inhibits Amyloid-β oligomers (AβOs) and decreases tau phosphorylation. Cu(II)GTSM also decreases the abundance of Amyloid-β trimers. Cu(II)GTSM is a potential

Cu(II)GTSM, a cell-permeable Cu-complex, significantly inhibits GSK3β. Cu(II)GTSM inhibits Amyloid-β oligomers (AβOs) and decreases tau phosphorylation. Cu(II)GTSM also decreases the abundance of Amyloid-β trimers. Cu(II)GTSM is a potential

Cu(II)GTSM, a cell-permeable Cu-complex, significantly inhibits GSK3β. Cu(II)GTSM inhibits Amyloid-β oligomers (AβOs) and decreases tau phosphorylation. Cu(II)GTSM also decreases the abundance of Amyloid-β trimers. Cu(II)GTSM is a potential

Cu(II)GTSM, a cell-permeable Cu-complex, significantly inhibits GSK3β. Cu(II)GTSM inhibits Amyloid-β oligomers (AβOs) and decreases tau phosphorylation. Cu(II)GTSM also decreases the abundance of Amyloid-β trimers. Cu(II)GTSM is a potential

Cu(II)GTSM, a cell-permeable Cu-complex, significantly inhibits GSK3β. Cu(II)GTSM inhibits Amyloid-β oligomers (AβOs) and decreases tau phosphorylation. Cu(II)GTSM also decreases the abundance of Amyloid-β trimers. Cu(II)GTSM is a potential