Potassium bitartrate/sodium bicarbonate

5-Iodo-Indirubin-3'-monoxime is a highly potent, cell-permeable, and reversible inhibitor of GSK-3β. 5-Iodo-Indirubin-3'-monoxime also inhibits Cdk1 and Cdk5 activities by binding to the ATP-binding pocket of these

Biochem/physiol Actions A-278637 is a selective opener of ATP-sensitive K (KATP) channels (EC 50 = 102 nM) that blocks contraction of bladder smooth muscle in both in vivo and in vitro assays. A-278637 is 15-fold more selective than

A-674563 is an Akt1 inhibitor with Ki of 11 nM in cell-free assays, modest potent to PKA and &gt:30-fold selective for Akt1 over PKC.A selective Akt1, PKA, and CDK2 inhibitor.

A-674563 is an Akt1 inhibitor with Ki of 11 nM in cell-free assays, modest potent to PKA and &gt:30-fold selective for Akt1 over PKC.A selective Akt1, PKA, and CDK2 inhibitor.

A-674563 is an Akt1 inhibitor with Ki of 11 nM in cell-free assays, modest potent to PKA and &gt:30-fold selective for Akt1 over PKC.A selective Akt1, PKA, and CDK2 inhibitor.

ABT-072 (potassium trihydrate) is an orally active and potent non-nucleoside HCV NS5B polymerase inhibitor (HCV GT1a EC 50 =1 nM: HCV GT1b EC 50 =0.3 nM)In VitroABT-072 (potassium trihydrate) is a non-nucleoside NS5B polymerase inhibitor with

ABT-072 (potassium trihydrate) is an orally active and potent non-nucleoside HCV NS5B polymerase inhibitor (HCV GT1a EC 50 =1 nM: HCV GT1b EC 50 =0.3 nM)In VitroABT-072 (potassium trihydrate) is a non-nucleoside NS5B polymerase inhibitor with

ABT-072 (potassium trihydrate) is an orally active and potent non-nucleoside HCV NS5B polymerase inhibitor (HCV GT1a EC 50 =1 nM: HCV GT1b EC 50 =0.3 nM)In VitroABT-072 (potassium trihydrate) is a non-nucleoside NS5B polymerase inhibitor with

ABT-072 (potassium trihydrate) is an orally active and potent non-nucleoside HCV NS5B polymerase inhibitor (HCV GT1a EC 50 =1 nM: HCV GT1b EC 50 =0.3 nM)In VitroABT-072 (potassium trihydrate) is a non-nucleoside NS5B polymerase inhibitor with

A selective activator of AMPK inhibits synthesis of fatty acids and sterols in hepatocytes and insulin-stimulated glucose uptake in adipocytes.

A selective activator of AMPK inhibits synthesis of fatty acids and sterols in hepatocytes and insulin-stimulated glucose uptake in adipocytes.

A selective activator of AMPK inhibits synthesis of fatty acids and sterols in hepatocytes and insulin-stimulated glucose uptake in adipocytes.