Potassium nitrate/sodium fluoride

A potential metabolite of fluprostenol when administered to animals a common minor impurity found in commercial preparations of the bulk drug compound.

A potential metabolite of fluprostenol when administered to animals a common minor impurity found in commercial preparations of the bulk drug compound.

A potential metabolite of fluprostenol when administered to animals a common minor impurity found in commercial preparations of the bulk drug compound.

2'-FLUORO-4'-NITROACETANILIDE-

[¹²⁵I]NKA (human, mouse, rat).

[¹²⁵I]NKA (human, mouse, rat).

InformationA-83-01 is a potent inhibitor ofTGF-β type I receptor (ALK5-TD)with IC50 of 12 nM. A-83-01 also inhibits the transcription induced byactivin/nodal type I receptor (ALK4-TD)andnodal type I receptor (ALK7-TD)with IC50 of 45 nM and 7.5 nM,

AG 538 is a potent, competitive inhibitor of IGF-IR (IGF-1 receptor kinase, IC|50|= 400 nM). Reaction is competitive with respect to IGF-1R substrate. Also inhibits the phosphorylation of PTK substrate poly(Glu,Tyr) by IGF-1R, IR, EGF-R, and Src

AG 538 is a potent, competitive inhibitor of IGF-IR (IGF-1 receptor kinase, IC|50|= 400 nM). Reaction is competitive with respect to IGF-1R substrate. Also inhibits the phosphorylation of PTK substrate poly(Glu,Tyr) by IGF-1R, IR, EGF-R, and Src

AG 538 is a potent, competitive inhibitor of IGF-IR (IGF-1 receptor kinase, IC|50|= 400 nM). Reaction is competitive with respect to IGF-1R substrate. Also inhibits the phosphorylation of PTK substrate poly(Glu,Tyr) by IGF-1R, IR, EGF-R, and Src

AG 538 is a potent, competitive inhibitor of IGF-IR (IGF-1 receptor kinase, IC|50|= 400 nM). Reaction is competitive with respect to IGF-1R substrate. Also inhibits the phosphorylation of PTK substrate poly(Glu,Tyr) by IGF-1R, IR, EGF-R, and Src

Anti-AK5 adenylate kinase 5, 100ul UN 1687 6.1 PG2