Cintazone or cinnopentazone

(5Z,2E)-CU-3 is a potent and selective inhibitor against the α-isozyme of DGK with an IC 50 value of 0.6 μM, competitively inhibits the affinity of DGKα for ATP with a K m value of 0.48 mM. (5Z,2E)-CU-3 targets the catalytic region, but not the

1-[6-fluoro-4-(4,4,5,5-tetramethyl-1,3,2-dioxaborolan-2-yl)indazol-1-yl]ethanone.

2-​Oxa-​6-​azaspiro[3.3]​heptane-​6-​ethanamine.

It has been used to explore deoxycholic acid (DCA) induced changes in cell signaling. DCA is a secondary bile acid implicated in numerous pathological conditions. It has also been used in the synthesis of (-)-curcumanolide A and (-)-curcumalactone

Anti-NCLN nicalin, 25ul UN 1687 6.1 PG2

Anti-NKD1 naked cuticle homolog 1 (Drosophila), 100ul UN 168

Anti-NKD1 naked cuticle homolog 1 (Drosophila), 25ul UN 1687

Anti-NKD1 naked cuticle homolog 1 (Drosophila), 25ul UN 1687

InformationApremilast (CC-10004) is a potent and orally activePDE4andTNF-αinhibitor withIC50of 74 nM and 77 nM, respectively.In vitroApremilast is more potent for inhibition of PDE4 compared with cAMP or cGMP hydrolysing enzymes from other PDE

c-Fms-IN-3 is a novel c-Fms kinase inhibitor with a potential as anti-inflammatory agent and antirheumatic agent.IC50&amp: Target:c-Fms.

C3a, along with C4a and C5a, is a 77 amino acid anaphylotoxin generated by proteolytic cleavage during activation of the complement pathway (Ember, 1997). The anaphylotoxins strongly promote inflammation by recruiting leukocytes, particularly

C5a is a proinflammatory peptide generated through activation of the complement system, and is more potent than the other anaphylatoxins, C4a and C3a, in activating peripheral blood leukocytes (Gerard &amp Gerard, 1994). It can actively