Nonsteroidal anti inflammatory glycolates

GSK717 is a potent, selective NOD2 (nucleotide-binding oligomerization domain 2) inhibitor. GSK717 inhibits muramyl dipeptide (MDP)-induced NOD2-mediated signaling, with an IC 50 of 400 nM for MDP-stimulated IL-8 secretion in HEK293/hNOD2 cells.In

GSK717 is a potent, selective NOD2 (nucleotide-binding oligomerization domain 2) inhibitor. GSK717 inhibits muramyl dipeptide (MDP)-induced NOD2-mediated signaling, with an IC 50 of 400 nM for MDP-stimulated IL-8 secretion in HEK293/hNOD2 cells.In

GSK717 is a potent, selective NOD2 (nucleotide-binding oligomerization domain 2) inhibitor. GSK717 inhibits muramyl dipeptide (MDP)-induced NOD2-mediated signaling, with an IC 50 of 400 nM for MDP-stimulated IL-8 secretion in HEK293/hNOD2 cells.In

GSK717 is a potent, selective NOD2 (nucleotide-binding oligomerization domain 2) inhibitor. GSK717 inhibits muramyl dipeptide (MDP)-induced NOD2-mediated signaling, with an IC 50 of 400 nM for MDP-stimulated IL-8 secretion in HEK293/hNOD2 cells.In

An allosteric inhibitor of oncogenic K-Ras(G12C).

A potent and selective EP4 receptor antagonist (Ki = 24 nM) reverses the anti-inflammatory action of PGE2 in LPS - activated human macrophages at 100 nM.

A potent and selective EP4 receptor antagonist (Ki = 24 nM) reverses the anti-inflammatory action of PGE2 in LPS - activated human macrophages at 100 nM.

A potent and selective EP4 receptor antagonist (Ki = 24 nM) reverses the anti-inflammatory action of PGE2 in LPS - activated human macrophages at 100 nM.

General description FP-Series: Fluorescein-Labeled Polymeric Probes These glycoconjugate probes are designed to reveal lectins, including selectins, directly on cell membranes, organelles, and in cytoplasm. The low fluorescein content ensures

It can delay and inhibit the production of excessive cell interleukin, thereby   inhibiting some unnecessary and inappropriate inflammation and glycosylation damage.Efficacy and Mechanism of ActionPalmitoyl tetrapeptide-7 (Rigin TM) (sequence:

It can delay and inhibit the production of excessive cell interleukin, thereby   inhibiting some unnecessary and inappropriate inflammation and glycosylation damage.Efficacy and Mechanism of ActionPalmitoyl tetrapeptide-7 (Rigin TM) (sequence:

It can delay and inhibit the production of excessive cell interleukin, thereby   inhibiting some unnecessary and inappropriate inflammation and glycosylation damage.Efficacy and Mechanism of ActionPalmitoyl tetrapeptide-7 (Rigin TM) (sequence: