Molecular Biology

Application A powerful ligand for classic cross-coupling reactions combined with the Buchwald Fourth Generation Palladacycle. Bench stable, soluble in most organic solvents.

Application A powerful ligand for classic cross-coupling reactions combined with the Buchwald Fourth Generation Palladacycle. Bench stable, soluble in most organic solvents.

rac-BINAP-Pd-G3 is a third generation (G3) Buchwald precatalyst. t is air, moisture and thermally-stable and is highly soluble in a wide range of common organic solvents.

rac-BINAP-Pd-G3 is a third generation (G3) Buchwald precatalyst. t is air, moisture and thermally-stable and is highly soluble in a wide range of common organic solvents.

Specificity Predicted to cross-react with all mammalian species Anti-Rac1, clone 23A8 (mouse monoclonal IgG2b): Human, mouse and rat, other species cross-reactivity unknown. Note: This antibody may be reused 3-5 times for immunoblot analysis.

Rac1/cdc42 Activation Magnetic Beads Pulldown Assay (Cat. No.

Recombinant, Pneumocystis carinii RAD51 fused at the N-terminus to a His&#8226Tag sequence and expressed in E. coli .

A cell-permeable triarylimidazole compound that acts as a reversible, ATP-competitive, and highly potent inhibitor of Raf kinase (IC 50 = 10 nM &amp K d = 2.4 nM for B-raf) with 7-, 30-, and 70-fold selectivity over p38&#945, GSK-3&#946, and

N-terminal GST-tagged recombinant human Raf-1 residues 306-end, containing the mutations Y340D and Y341D Product Source: Expressed by baculovirus in Sf21 insect cells Biochem/physiol Actions Protein Target: c-RAF Target Sub-Family: TKL Packaging

N-terminal GST-tagged recombinant human Raf-1 residues 306-end, containing the mutations Y340D and Y341D Product Source: Expressed by baculovirus in Sf21 insect cells Biochem/physiol Actions Protein Target: c-RAF Target Sub-Family: TKL Packaging

A potent, cell-permeable, reversible, and ATP-competitive cRAF1 kinase inhibitor (IC 50 = 9 nM). Shows &#8805100-fold selectivity for Raf kinase versus Cdk1, Cdk2, c-Src, ERK2, MEK, p38, Tie2, VEGFR2, and c-Fms.

A cell-permeable compound that specifically, reversibly, and stoichiometrically (1:1) binds to the GDP-bound Ral A/B in an allosteric manner (K d = 7.7 &#181M) and blocks its activation.